BRAF and melanoma: Pathway analysis of transcripts showed that pathways associated with cytokine–cytokine receptor interaction, ECM receptor interaction, and neuroactive ligand receptor interaction were similarly enriched in patient-derived metastatic BRAF mutant Mel 14-108 melanoma as in M14 and A2058 models, whereas pathways associated with the cell cycle, DNA replication, Fanconi anemia, and DNA repair pathways were enriched in wild-type BRAF Mel 14-089 patient-derived melanoma, revealing distinct regulations by mutant and wild-type BRAF.