Although increased levels of STRAP have been correlated with cancer progression, its homozygous deletion resulted in mouse embryonic lethality and morphological defects, causing death at days E9.5 and E10.5 due to defects in angiogenesis, cardiogenesis, somitogenesis, and neural tube closure, suggesting its indispensable functions in embryonic development [16,17,18]. The gene discussed is STRAP; the disease is cancer.