ETV4 and neoplasm: It was recently reported that increased ORC6 expression is closely associated with tumor progression and an unfavorable prognosis in NSCLC patients.[63] We found that depleting ORC6 significantly inhibited NSCLC cell proliferation, EdU incorporation, and S phase progression and reversed the effects of ETV4 on cell proliferation and EdU incorporation, which indicated the essential role of ORC6 in ETV4‐regulated tumor cell proliferation and replication.