No prior studies have investigated BAFF/APRIL inhibition in the experimental model of autoimmune myasthenia gravis (EAMG), a well-characterized model used to study the mechanisms involved in MG (19, 20), but exploration of the potential for BAFF/APRIL in MG patients may be enhanced by a comparative study in EAMG vs. other mechanisms of clinical interest. This evidence concerns the gene TNFSF13 and myasthenia gravis.