ZNF793, which is typically unmethylated in EBV GC, is crucial for maintaining cell proliferation, cell migration, cell invasion, and tumor growth in GCs with expression of ZNF793. Knockout experiments demonstrated that the absence of ZNF793 significantly suppressed these stemness- and oncogenesis-related processes, whereas its reintroduction restored these processes. This evidence concerns the gene ZNF793 and gastric cancer.