AKT1 and Glucose intolerance: Dietary Se‐deficiency increased fasting insulin (54%; Figure 4b) and glucose (33%; Figure 4c) concentrations in postmortem sera, induced glucose intolerance (38%; Figure 4d) and insulin resistance (31%; Figure 4e), and decreased (27%–56%) baseline levels of skeletal muscle p‐AKT S473 and T308, as well as liver p‐AKT S473 (Figures 4f,g and S7).