A previous study reported that macrophage‐fibroblast crosstalk plays an important role in the pathophysiology of cardiac inflammation and fibrosis.[27] To further investigate the role of PPT1‐expressing macrophages in myocardial fibrosis, conditioned medium (CM) from M1 macrophages was harvested and added to cardiac fibroblasts (Figure S8A, Supporting Information). This evidence concerns the gene PPT1 and inflammation.