ADH5 and infection: Moreover, we predicted the key interaction sites between PcRD18 and GSNOR (Figure 3) and mutation of these sites in PcRD18 (PcRD18‐IM) resulted in loss of its ability to target and suppress GSNOR, as well as its ability to disturb the NO homeostasis, inhibit the ROS burst and promote infection (Figure 7D–I).