They identified distinct metabolic profiles connected to postpartum depressive symptoms, indicating that disruptions in lipid metabolism and immune-related metabolites may play a key role in the development of PPD.83 Furthermore, another study by Min et al84 found a positive correlation between elevated Th17 cells and related cytokine IL-17A with both PPD and anxiety, which further supports the role of Th17-mediated inflammation in these conditions. The gene discussed is IL17A; the disease is progressive pseudorheumatoid arthropathy of childhood.