EGFR and nasopharyngeal carcinoma: Our findings revealed that DDAH1 interacts with EGFR in vitro, whose activation is associated with poor survival in many tumors and is highly expressed in most NPC tissues.[] Furthermore, we found that overexpression of DDAH1 enhanced cisplatin resistance and made an increase in p‐EGFR by comparing with control group in Figure 7B in vivo, which consolidates the conclusions in vitro.