The common processes included physiological pathways specifically related to the physiology of viral infection, including: 'innate immune response,' 'negative regulation of viral genome replication,' 'regulation of viral genome replication,' 'response to interferon-alpha,' 'response to interferon-beta,' 'response to type I interferon' and 'type I interferon signaling pathway.' These results show that the main cellular processes disturbed by virus-related methylation changes in infected cells are those essential for the infection progression and general host response to the infection. The gene discussed is IFNB1; the disease is viral infectious disease.