Consistent with our transcriptomic findings, LGALS9 treatment upregulated the expression of genes involved in cholesterol metabolism (NPC2, APOA) and PPAR signalling (SCD, PLIN2, FABP1) (Figure 4H), suggesting that LGALS9‐P4HB interaction modulates lipid metabolism in gastric cancer cells. This evidence concerns the gene LGALS9 and gastric cancer.