We found that the changed genes were involved in response to corticosteroids, response to oxidative stress, regulation of inflammatory response, collagen-containing ECM, immunoglobulin complex, circulation, clathrin-coated vesicle membrane, cytokine activity, signaling receptor activator activity, receptor ligand activity, rheumatoid arthritis, and NF-κB signaling. The gene discussed is NFKB1; the disease is rheumatoid arthritis.