TGFB1 and non-small cell lung carcinoma: TAMs in the interstitium of NSCLC are predominantly of the M2 phenotype (CD68+TGF‐β1+), which is significantly higher than that in paracancerous tissues, and the M2 phenotype inhibits antitumor immunity by secreting factors such as IL‐10 and TGF‐β, promoting angiogenesis and stromal remodeling [165].