HSP90B1, EEF2, and ANXA1 are involved in cellular stress responses and have been reported to be linked to gliosis-associated neuroinflammation [37–39].To predict the network-based functional implications of proteins modulated by GFAP mAb treatment, we performed a GENEMANIA bioinformatic analysis on genes corresponding to the markers identified in our Western blot studies: GFAP, CASP1, NF-κB1, MAPK14, TLR4, NLRP3, and GSDMD. Here, NFKB1 is linked to Gliosis.