While tumor epithelial cells were captured from almost all tumors, the capture rate for stromal and immune cells was highly variable among the specimens (Supplementary Fig. 2C) per our annotation based on graphical clustering patterns and canonical cell-type specific markers for tumor epithelial/urothelial cells (EPCAM, KRT7), immune cells (PTPRC), stromal cells (DCN, ACTA2), and endothelial cells (SELE) (Supplementary Fig. 3A). The gene discussed is EPCAM; the disease is neoplasm.