Bold values indicate critical abnormalities.RBS and HbA1c confirm severe hyperglycemia and poor long-term glycemic control. pH, low HCO3, and low PaCO2 reflect high anion gap metabolic acidosis with respiratory compensation—hallmarks of DKA. Positive GAD autoantibodies confirm autoimmune etiology consistent with T1DM. Urine glucose (4+) and ketones (2+) support the presence of glycosuria and ketonuria typical of DKA. The gene discussed is GAD1; the disease is Hyperglycemia.