CPT1A has also been found to have functions beyond fatty acid oxidation, as it has been implicated in antioxidant capacity and ferroptosis resistance in cancer cells.64,65 Given that ferroptosis is an iron-dependent form of cell death triggered by lipid peroxidation, CPT1A may contribute to redox homeostasis in a manner that is not fully restored by NAC alone. This evidence concerns the gene CPT1A and cancer.