TGFB1 and pulmonary fibrosis: Disruption of in vivo lipidhomeostasis underlies age-related diseases, and glyceryl triacetate attenuateshippocampal neuronal senescence and improves cognitive function in aged rats (37, 38).By contrast, m6A-modified genes predominantly participated in oncogenic signalingpathways, including the TGF-β cascade, which promotes ROS generation andsenescent cell accumulation in pulmonary fibrosis (39, 40).