The histopathological examinations revealed substantial damage to the kidney and liver tissues of the EAC-bearing group that could be correlated to the accumulation of hemorrhagic ascitic fluid, tumor metastasis, and invasion of tumor cells.142–144 Consistent with our biochemical analysis, our findings revealed a decrease in the serum ALB and total protein levels and an increase in the AST, ALT, urea, and creatinine levels in the EAC-induced group, suggesting liver and kidney dysfunctions. The gene discussed is GPT; the disease is neoplasm.