Along these lines, Giri et al. (2023) reported that harmine–in combination with the FOXP3 activator Kaempferol–enhanced NFATC1/FOXP3-mediated Treg’s suppressive capacity in vitiligo models, leading to reduced CD8+ and CD4+ T-cell proliferation, reduced IFN-γ production, and increased melanocyte survival and proliferation. This evidence concerns the gene FOXP3 and vitiligo.