Glucose exerts an indispensable role in cellular function, as it turns out that levels of myofibroblast glycolytic metabolism are increased in IPF, and elevated glycolysis stabilizes hypoxia‐inducible factor 1 alpha, an essential ingredient for myofibroblast differentiation [31], which is in agreement with our results: higher levels of glucose are a hazardous condition for IPF. This evidence concerns the gene HIF1A and idiopathic pulmonary fibrosis.