For example, a coumarin–dithiocarbamate hybrid showed inhibitory effects on both acetylcholinesterase (AChE) and monoaminoxidase B (MAO B) and reversed scopolamine-induced memory deficits in mice, underlining the potential of this structural class for the treatment of neurodegenerative diseases, such as Alzheimer’s disease (AD) [9]. Here, MAOB is linked to early-onset autosomal dominant Alzheimer disease.