These results, together with the high expression of hsa-miR-182 in tumor samples compared to non-tumor samples (Figure 2A), allow us to suggest that although all three miRNAs described in this study show a high binding probability to TP53INP1, hsa-miR-182 may exert a stronger influence on the regulation of its expression in the context of this disease. The gene discussed is TP53INP1; the disease is neoplasm.