Molecular analyses have revealed the presence of SARS-CoV-2 proteins and injury markers, such as glial fibrillary acidic protein (GFAP) and agrin, in astrocyte- and neuron-derived exosomes (ADEVs and NDEVs), even in individuals with mild acute COVID-19, indicating persistent cellular dysfunction [51,58,69,79]. The gene discussed is GFAP; the disease is COVID-19.