The μ-bcr breakpoints occur beyond exon 19 (e19) of the BCR gene, leading to the larger p230 oncoprotein, which is rare and associated with neutrophilic CML and characterized by predominant neutrophilic maturation and/or thrombocytosis [7]. The gene discussed is GOLGA4; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.