This can occur through multiple mechanisms, including expression of the dendritic cells chemoattractant XCL1, which increases tumour antigenicity to prime T cell responses [7], and secretion of T-cell recruiting chemokines such as CCL2, CCL5, CXCL9, and CXCL10 to attract cytotoxic CD8+ T cells to tumour sites [8]. The gene discussed is CD8A; the disease is neoplasm.