This study compared the extent of airway remodeling in different immunological endotypes of asthma.<h4>Methods</h4>Severe asthma patients (n = 30) were stratified based on bronchial expression of T2 (e.g., CST1) and T3 (e.g., IL17A) immunity genes as T2-high, T3-high, or low-inflammatory. This evidence concerns the gene IL17A and asthma.