In this context, it might be interesting to note that perturbed expression of GLT1 (EAAT2, SLC1A2), the principal transporter mediating about 95% of the transport and clearance of l-glutamate (and potentially l- and d-aspartate) from the extracellular space in brain [286] could also be involved in the hypothetical d-aspartate dysfunction in schizophrenia. Here, SLC1A2 is linked to schizophrenia.