CX3CR1 and infection: In agreement with these findings, we observed that Cx3cr1 deficiency and EGFP knock-in in Cx3cr1/EGFP mice did not alter the incubation time to disease following infection with mouse-adapted SSLOW or 22L prion strains, compared to wild-type (WT; C57BL/6J) controls (Figure S1a–c).