Kennedy et al. (2021) investigated whether the transient changes in circulating monocytes—specifically, the expression of CCR2 and CX3CR1 observed 1 to 3 months after infection—resulted from the highest levels emigrating toward inflamed or damaged tissues, given that CCL2 was linked to the recruitment of monocytes to the lungs during infection [56]. The gene discussed is CX3CR1; the disease is infection.