Consistent with the hypothesis that dry season asymptomatic carriers have an increased ability to restrict PfEMP1s, we detected higher surface recognition of VSAs (Fig 5B), and a broader recognition of CD36-, EPCR- and other group A PfEMP1-binders (Fig 5C) in plasma of the dry season asymptomatic infections than in those of clinical malaria cases. Here, CD36 is linked to malaria.