The research findings presented in this study shed light on the role of Snap25 protein or snap25 gene expressions in the development of ADHD, which are consistent with previous studies in animal models and observations from mutations in Snap25 in humans, indicating that this protein plays a crucial role in inducing ADHD-like symptoms in animal models [28, 29] and ADHD in humans [2, 3, 5, 26, 27]. This evidence concerns the gene SNAP25 and attention deficit-hyperactivity disorder.