On the seventh day post-stroke, bacterial load in lung tissue significantly increases, and the expression levels of inflammatory cytokines such as Interleukin-1 beta (IL-1β), Interleukin-6 (IL-6), MCP-1, MIP-1α, and Tumor Necrosis Factor-alpha (TNF-α) show significant changes, indicating immune system dysregulation and sustained inflammation. Here, IL1B is linked to stroke disorder.