Most studies have implicated EBV‐driven oncogenic mechanisms, including the activation of the NF‐κB pathway and upregulation of BCL2[9, 10] in tumor cells, alongside tumor environment (TME) modulation, such as M2‐type tumor‐associated macrophages (TAMs)[11] polarization and PD‐L1 upregulation.[12] However, few studies have focused on the role of the peripheral nervous system, which also plays a crucial role in cancer development and progression. Here, BCL2 is linked to neoplasm.