AKT1 and cervical cancer: Another study revealed that FA (4–20 μM) could stimulate apoptosis in HeLa and CaSki cells by increasing cytotoxicity, pro-caspase-3, pro-caspase-8, and pro-caspase-9, PARP, Bax, ROS, DNA condensation, and decreasing Bcl-2 and Mcl-1, AKT, PI3K phosphorylation in cervical cancer [99].