These included tumor suppressors down-regulated and/or inactivated by AR, including CD24 [41], GATA binding protein 3 (GATA3) [34], p53 [31], and UDP-glucuronosyltransferase 1A (UGT1A) [42], as well as oncogenic molecules up-regulated and/or activated by AR, including ATF2 [43], c-fos [44], ETS transcription factor ELK1 [44], and nuclear factor-κB (NF-κB) [45]. This evidence concerns the gene AR and neoplasm.