CPT1A and lung cancer: Recent studies have demonstrated that c‐Myc activation in lung cancer cells upregulates CPT1A and further enhances the antioxidant capacity of cells through the Nrf2 pathway.[30] The c‐DTP cells exhibited a more pronounced increase in fatty acid uptake and OXPHOS compared to DTP cells, mostly likely due to activation of the c‐MYC pathway and increased CPT1A expression.