EEF2 and Alzheimer disease: Further supporting nMAβ42‐induced TyrRS depletion (Figure S1B,C), treatment with nMAβ42 increased phosphorylation of eEF2 (Figure S1E), providing a potential molecular basis for nMAβ42‐induced TyrRS depletion via neuronal protein synthesis inhibition and deficits in recall after contextual fear memory in 8‐to 12‐month‐old AD female model mice [98].