Eosinophil activation by platelets has shown to occur during type II immune responses in diseases like asthma.30, 31, 32 The activation of eosinophil β1-integrin through P-selectin takes place in asthma, leading to the swift migration of platelet-eosinophil complexes into the lungs, particularly in more severe cases.31 The gene discussed is SELP; the disease is asthma.