Figure 3 highlights various important related pathways implicated in the pathogenesis of glioma, including phosphatidylinositol-3’-kinase (PI3K)/Akt, mammalian target of rapamycin (mTOR), and Janus kinase (JAK)/signal transducer and activator of transcription (STAT) pathways. Here, SOAT1 is linked to glioma.