RPS3A has been reported to promote PPARγ expression[33] and the transport of transcription factors such as NF‐κB from the cytoplasm to the nucleus.[34] Immunoprecipitation analysis confirmed that p53 and RPS3A indeed interact on LDs purified from tumor cells (Figure 5D; Figure S6C, Supporting Information). The gene discussed is NFKB1; the disease is neoplasm.