Growing evidence indicates that epigenetic modifications play important roles in tumorigenesis and cancer progression.[35] As the most abundant post‐transcriptional epigenetic modification of RNAs in eukaryotes, m6A has been demonstrated to exert critical molecular functions in modulating lncRNAs.[36, 37] m6A is installed by methyltransferases (known as “writers”), which catalyze m6A modification via methyltransferase domains.[38] We identified that the writer METTL14 promoted the m6A modification in LINC01094. Here, METTL14 is linked to cancer.