GLUT1, a glucose transporter, plays a pivotal role in tumor metabolism and significantly accelerates cancer cell metabolism under both aerobic and anaerobic conditions.[42] HK2, as the rate‐limiting enzyme in glycolysis, is directly involved in the metabolic reprogramming of tumor cells.[43] Our research identified two mechanisms by which lncRNAs regulate GLUT1 and HK2 expression. The gene discussed is SLC2A1; the disease is cancer.