However, it is important to note that we did not find that all vulnerable neuronal subtypes were associated with intraAβ accumulation (e.g., ADARB1+ neurons) and found that neuronal populations showing low levels of intraAβ accumulation were not vulnerable to loss with AD progression, e.g., calretinin+, CALB1+, and PVALB+ inhibitory neurons. This evidence concerns the gene PVALB and Alzheimer disease.