FGFR3 and cancer: Although we cannot rule out the possibility that PARP1 Y158 residue can be phosphorylated by other FGFR members in various cancer types, we found that Y158 phosphorylation is predicted to be mediated only by FGFR3 among the FGFR family using the algorithm of group-based phosphorylation site predicting analysis (http://gps.biocuckoo.cn/) (13).