The fibroblast growth factor 2 (FGF2) derived from nasopharyngeal carcinoma cells (NPC) was shown to induce pericyte-specific expression of CXCL14, which in turn promoted recruitment of TAMs and their polarization to an M2-like phenotype; and lung metastasis induced by FGF2 was reduced following inhibition of TAMs using clodronate liposomes (67). The gene discussed is FGF2; the disease is nasopharyngeal carcinoma.