CD36 and atrial fibrillation: This apparently discordant finding is consistent with the results of two proteomic studies that reported the upregulation of proteins involved in fatty acid uptake into the mitochondria and β-oxidation, such as fatty acid translocase (FAT/CD36), (14)C-palmitic acid and CD36, which resulted in an apparent increased activity of the oxidative phosphorylation pathway in the AF groups [101,102].