Additionally, impaired renal function can lead to increased biomarker levels (e.g., cTn [72] or BNP and NT-proBNP [73]), and many other diseases or circumstances interfere with postmortem biomarker levels (e.g., ventricular arrhythmia increases Tn [74] and BNP levels [75]; trauma can increase Tn levels [76]; sepsis can be associated with elevated Tn [77, 78] and NT-proBNP [78] levels; electrocution elevates cTn levels [79]; asphyxia can increase cTnI and myo levels [80]; and cardiopulmonary resuscitation can lead to elevated markers of myocardial damage [18, 81–83]). The gene discussed is NPPB; the disease is Ventricular arrhythmia.