Because IL-29 is the only IFN-λ secreted by AECs infected with influenza A virus (IAV), and IP-10 is an IL-29-induced ISG molecule associated with viral clearance (43), we explored whether the divergent susceptibility in infection of different H1N1 viruses was due to the different secretion of IL-29 and IP-10. The gene discussed is IFNL1; the disease is infection.